Facilitator Questions

Why do cells have such a wide array of pinocytic mechanisms?

It was mentioned in the lecture that several viruses use more than one endocytic mechanism. How can a virus do this?

In the lecture two mechanisms were discussed for the penetration of non-enveloped viruses, endosome lysis and pore formation. Which mechanism do you think is more advantageous and why?

What mechanisms could a virus use to associate with and move along microtubules?

What advantages could using viruses to study endocytosis have over the use of other classical endocytosis ligands?

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